Acne Treatment Medication

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Some medications are known to cause acne. Some cortisones, few anti-tuberculosis medications and some anti-epileptic and anti-seizure medications can cause acne. Also medications that include anabolic steroids, and lithium and iodine-based medications.
Hormone medications like contraceptive solutions and older oral contraceptives may make acne worse. Other medications known to exacerbate acne include some antidepressants, and cyclosporin.
Thyroid Medications: prescribed to activate the thyroid gland in patients with low thyroid function. Acne is a side effect.
Disulfuram - prescribed for alcoholic patients trying to achieve sobriety. Constant use can trigger acne.
Immunosuppressants - prescribed to suppress the immune system; primarily used to prevent organ rejection in patients enduring transplants. Immune suppression permits bacteria to grow, including the bacteria that causes acne, Proprionibacterium acnes.
Oral Vitamin A: Retinoids (derivatives of vitamin A) are applied locally and orally to treat acne under medical supervision. Vitamin A does not treat acne. If you take excessive vitamin A, hoping that it will treat acne, your health may become worse. Keep in mind that Vitamin A in excess quantity can have negative consequences on the body.
Hereditary: Acne can be inherited. If your parents had acne, you may be more prone to it.
Hormonal Changes: Hormonal changes bring on acne. The hormone androgen is responsible for excess secretion of sebum. Women can suffer acne outbreaks during menstruation and pregnancy.
Acne-Like Ailments: Some other conditions such as folliculitis may appear like acne. There are several other conditions that may look like acne. Some of them are rosacea, perioral dermatitis, keratosis pilaris, etc. Always ask a doctor instead of trying self-treatment.
Usual concerns about treating acne
Excessive sebum secretion: At puberty, increasing levels of androgens, the major sebotrophic hormones, begin to drive an increase in sebum secretion. However, while androgenic production is important in the pathogenesis of acne, the average acne patient does not have significant endocrine abnormalities. Hormonal therapy is not indicated in the initial treatment of mild to moderate acne, but females who require oral contraception may be candidates for anti-androgen therapy early in the application of treatment.
Aberrant desquamation of the follicular epithelium: In acne, keratinocytes hyperproliferate and accumulate within the sebaceous follicle. As these aberrantly desquamated cells accumulate in the sebaceous follicle, they produce microcomedo development. The microcomedo is the initiator to all acne blemishes and is present in 80% of acne papules but is imperceptible to the unaided eye. However, as the already blocked follicle begins to fill with lipids, bacteria and cell fragments, the microcomedo changes to open or closed comedones (blackheads and whiteheads, respectively), both of which are non-inflammatory lesions. If P. acnes grows, inflammatory promoters are produced and inflammatory papules and pustules appear.
Microbial proliferation: The microenvironment of the follicle in acne is prone to colonization with P. acnes. This produces inflammation and the production of the noticeable papules and pustules with which acne patients commonly present to dermatologists.
Inflammation: Inflammation in acne happens as a consequence of humoral and cellular defensive responses to P. acnes proliferation.

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